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Advanced MASH-related liver fibrosis is typically the result of a chronic, sustained inflammatory response involving multiple factors, characterized by abnormal deposition of type I collagen in the extracellular matrix (ECM), with the activation of HSCs being a key driver of liver fibrosis (Chan et al

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Depletion of mitochondrial glutathione has been associated with increased neuronal injury and has been implicated in the pathogenesis of several neurodegenerative diseases, including Alzheimers, Parkinsons, and multiple sclerosis