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Panretinal laser photocoagulation therapy is a common strategy for treating high-risk PDR and some severe nonproliferative diabetic retinopathy (NPDR) since evidence shows that it reduces the risk of severe vision loss from PDR in patients with high-risk PDR and some severe NPDR

doi: 10.1093/brain/awac007 43

Akt is deactivated and Bcl-2-associated death promoter (BAD) phosphorylation is lost in one pathway due to a stalled survival pathway, whereas Fas cell surface death receptor (FAS) is upregulated through stimulation of c-Jun-N-terminal kinase, which activates caspase-8, phosphorylates Fas-associated protein with death domain (FADD), and truncates BH3-interacting domain death agonist (BID)