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The effect was attributed to microbial metabolites that promoted a more youthful systemic environment (for example, boosting the production of certain short-chain fatty acids and reducing pro-inflammatory cytokines) [121]

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Notably, the mechanism of human GSTO1-1 may be that the backbone amide group of Phe34 donates a hydrogen bond to the sulphur atom of the H-site bound GSH 18

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