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We demonstrate higher CSF-corrected GSH levels with normal aging, suggesting a GSH compensatory response to increased oxidative stress with older age

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Although our cerebral GSH results do not support the redox hypothesis of autism in the brain, previous results of decreased GSH in the plasma of autistic children may help explain some other inflammatory symptoms that autistic children experience (e.g

N-Acetylcystein liefert den Baustein fr die krpereigene Glutathion-Produktion, whrend Zink, Selen und B-Vitamine antioxidative Enzyme aktivieren und den Methylierungszyklus untersttzen