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The DNMT1 protein faithfully maintains the pattern of methylated CGs that would otherwise become lost through cell divisions, whereas the de novo methyltransferases, DNMT3a/b, can also catalyze non-CG methylation (Suetake et al., 2003

The convergence of these mechanistic findings on angiogenesis, fibroblast behavior, oxidative stress, and the NO system provides a plausible framework for the in vivo tendon, ligament, gastric-mucosal, and ischemia findings reported in rat studies

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