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However, direct human efficacy data remain relatively limited, a concern FDA has consistently highlighted

Current therapeutic approaches, such as disease-modifying anti-rheumatic drugs (DMARDs) and biologics, are limited by infection risks, poor tissue specificity, and suboptimal long-term efficacy, underscoring the need for novel therapies

What we do not know Several questions remain unanswered: Does prolonged amylin receptor agonism produce any adaptive changes in receptor expression or sensitivity that could alter efficacy or safety over years

Targeting AKT signaling sensitizes cancer to cellular immunotherapy