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10.1023/A:1005592401947

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Adaptive immune responses, mediated by T and B cells, produce autoantibodies, including anti-citrullinated protein antibodies (ACPAs) and rheumatoid factor (RF), forming immune complexes that exacerbate synovial damage (13)

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1 G37R mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1