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Unfortunately, the more specific targets (1, 2) for these BPC 157 effects remained outside of the present investigation, but we should assume that in few hours period such a high paracetamol over-dose may be able also to reflect the disturbances that would otherwise require much longer period

in enteric nerve fibers/terminals and glia within the myenteric submucosal plexuses (inguinea pigs, mice), suggesting a role in the regulation of ileal contractility [259]

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the failure of these treatments to restore function and slow functional disease progression can be traced to their inability to correct the underlying cause of the dysfunction- diminished synaptic connectivity among neurons in the striatum and neuronal loss in the substantia nigra (SN)