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It is this dynamic that has made measurement of serum ceruloplasmin the primary screening test for Wilsons.16 Ultimately, the excess retained copper in the hepatocytes of Wilsons patients leads to toxic injury to the cell and eventually spillage of copper into the circulation, where it may be deposited in other organs such as the brain, cornea, and kidneys

It is known that the cupric ions bound to CP are not exchangeable 2 , which seriously complicates determination of the Cu(II)-binding affinity of CP

Arch Toxicol Suppl 1980

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