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Frczyk, Ternary Cu(II) Complex with GHK Peptide and Cis-Urocanic Acid as a Potential Physiologically Functional Copper Chelate, MDPI AG, Aug

Mechanistic studies indicate that AKT-mTOR signaling can reduce cytoplasmic malonyl-CoA levels by inhibiting ACACA, thereby influencing CPT1 activity and promoting fatty acid entry into mitochondria for -oxidation [173, 174]

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