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Nature 458 , 11311135 (2009)

This dual preservation of both NAD+ and methylation resources represents a unique mechanism among metabolic modulators, positioning 5-amino-1MQ as a bridge between energy metabolism and epigenetic regulation

By selectively inhibiting NNMT an enzyme central to energy regulation and fat metabolism this compound has demonstrated, in preclinical studies, the capacity to elevate intracellular NAD and SAM levels, suppress lipid accumulation in adipocytes, and reduce white adipose tissue mass in diet-induced obese mouse models