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The high amount of released glutamate causes decreased concentration of gamma-aminobutyric acid (GABA) confirmed by MRI localized spectroscopy of brain metabolites

The related modulatory mechanisms namely regulating Hadhb-mediated FAO, the Sirt1/NF-kB signaling pathway, and mTORC2/PPAR-g signaling and other pathways are involved [72, 73]

There are sex-based differences in morbidity, rupture risk, and prognosis in patients ( GSTA4 methylation levels than healthy men

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