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c-MET has been an important target against HNSCC because of its involvement in metabolic dysregulation, tumor-microenvirment and immune modulation

doi:10.3109/14756366.2010.526769

Furthermore, comparative studies can help support organized research methodologies and documented evaluation procedures

Targeted homing of CCR2-overexpressing mesenchymal stromal cells to ischemic brain enhances post-stroke recovery partially through PRDX4-mediated blood-brain barrier preservation