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Oxidation of pyridine nucleotides during Fas- and ceramide-induced apoptosis in Jurkat cells: Correlation with changes in mitochondria, glutathione depletion, intracellular acidification and caspase 3 activation

Toxins 11 (1), 18

Adverse reactions to sulphonamide and sulphonamide-trimethoprim antimicrobials: clinical syndromes and pathogenesis

After exceeding the capacity threshold of liver cells, they are damaged, and copper is released into the bloodstream, where it remains unbound with ceruloplasmin, (CPN) and in this form it is very toxic, and it also easily accumulates in other organs, e.g., in the intestines, brain, kidneys, and cornea [1,2,3]