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In an effort to bring cellular clarity to our analyses, and to investigate UMNs direct response to treatment, we took advantage of the prpTDP-43 A315T -UeGFP mice, a CSMN reporter line in which diseased UMNs in mice that have TDP-43 pathology retain their fluorescence both in vivo and in vitro

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24 h later, 20 l medium with compounds at 5X of the desired concentration was added to the well for 24 h

Br J Pharmacol 178(5):11821199 Guo J, Wang R, Min F (2022) Ginsenoside Rg1 ameliorates sepsis-induced acute kidney injury by inhibiting ferroptosis in renal tubular epithelial cells