While not specifically studied for bone healing, its mechanism of reducing NF-kB activation could theoretically support the transition from inflammatory to regenerative phases
this allows for site-specific oxidative damage to the plasma membrane, causing cell lysis
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doi: 10.3389/fnins.2020.591177 Received 03 August 2020 Accepted 12 October 2020 Published 28 October 2020 Volume 14 - 2020 Edited by Paulo Henrique Rosado-de-Castro, DOr Institute for Research and Education (IDOR), Brazil Reviewed by Ming Zhang, Kunming Medical University, China