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Recent research has highlighted the involvement of glycine in neuroprotective mechanisms during cerebral ischemia
Critical illness is defined as a condition of oxidative stress, hyper-inflammation, and impaired mitochondrial or immune system function.[6] A growing body of evidence suggests that oxidative stress contributes to the development of critical illness complications, including multiple organ failure and SIRS [7]
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26, the CRE, BUN levels and survival percentage of AKI mice treated with Cu 5.4 O USNPs (at 2 g kg 1 concentration) were similar to those of AKI mice treated with 160 mg kg 1 of NAC, which was also in accordance with the histological analysis of renal tissues
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