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Even the gold standard anti-ferroptotic drug liproxstatin, previously thought to exclusively exert protection via directly trapping free radicals [17] , has recently been shown to prevent ferroptosis by inactivating iron in the lysosome [18]

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Beyond direct receptor activation, research has reported upregulation of growth hormone receptor expression in tendon fibroblasts following BPC-157 exposure, alongside enhanced cell migration and survival in tissue-repair models (PMID 21030672)