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2020;34:e22420

The toxicity of -synuclein is also responsible for impaired endoplasmic reticulum affecting also closely associated and functionally related Golgi apparatus, aggregation of anti-apoptotic proteins, and formation of pores in cellular membranes (Cooper et al

This is also why GHK-Cu appears frequently in longevity-adjacent research alongside other compounds studied for age-related cellular changes, since a quantifiable, age-correlated decline gives researchers a concrete variable to test against rather than a purely theoretical one