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In MS, GSH metabolism is impaired in regions of the CNS that have been demyelinated and correlates with mitochondrial dysfunction and axonal injury ( 2 Mechanisms of redox regulation in the CNS 2.1 GSH as an antioxidant in the brain The brain, being one of the most metabolically active organs in the body, requires a large share of oxygen relative to its mass

Thus, we assume that the lower concentrations of H 2 O 2 (up to 100 mgl 1 ) may cause the stimulative stress response, and increased cell replication observed as increased number of colonies, e.g., for 50 or 100 mgl 1 at 4 h of the exposure

Activation of egfr and Erbb2 by helicobacter pylori results in survival of gastric epithelial cells with DNA damage