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Identifying a sensory gating deficit in ME/CFS may help to identify the core pathology as the same neuroinflammatory cycle identified in other conditions and better insights (and treatment options) might come from those other conditions that share the same basic pathophysiology but where the pathological process is expressed as a different constellation of symptoms, perhaps due to individual variations in genetic predisposition, developmental stage, environmental stressors and perhaps gender

In addition to reprogramming techniques, emerging rejuvenation modalities include miRNA-loaded exosomes, telomere-activating small molecules, and stem cell transplantation, each of which has the potential to reverse aging phenotypes through distinct mechanisms

Delivery of siRNA to the mouse brain by systemic injection of targeted exosomes

It also appears to exert powerful anti-inflammatory effects and modulate the nitric oxide (NO) pathway, further contributing to a healing-conducive environment