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After a 12-week trial with modified KD in 21 AD patients, daily function and quality of life improved (148)

153 As a compensatory mechanism, IAPP may activate HIF-1/6-phosphofructo-2-kinase/fructose-2,6-biphosphatase 3 (PFKFB3) to protect against oligomer toxicity, thus delaying the loss of cells but at the cost of impairing cell function

On the other hand, the plasma membrane glutathione efflux can be facilitated by specifically or ubiquitously expressed membrane proteins and anion channels such as multidrug resistance-associated proteins (MRP1-5), Cystic Fibrosis Transmembrane Conductance Regulator (CFTR), Arginine/Ornithine Transport ATP-binding Proteins (OATP 1,2), and ATP-Binding Cassette superfamily G member 2 (ABCG2) [19]

Br J Haematol 162(5):631638