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Conclusion Research on BPC-157 and GHK-Cu cream has demonstrated promising preclinical findings in tissue repair and recovery
mTORC1 directly inhibits AMPK to promote cell proliferation under nutrient stress
3.3.3 Altered fatty acid metabolism affecting follicular growth HFD disrupts ovarian fatty acid metabolism by upregulating fatty acid uptake and trafficking (increased CD36, FABP expression), impairing -oxidation (reduced PPAR targets, CPT1, ACOX), and promoting lipid storage via PPAR-mediated FSP27/CIDE-C upregulation, leading to ectopic lipid accumulation, lipotoxicity, and oxidative stress compromising folliculogenesis at multiple stages ( In HFD-fed rodents, increased expression of fatty acid transporters (e.g., CD36) and lipid droplet-associated proteins (e.g., FSP27) contributes to abnormal lipid accumulation and oxidative stress in ovarian cells ( Dysregulation of key nutrient-sensing and longevity-related regulators, such as SIRT1 and FOXO3, has also been observed under HFD conditions
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