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Activin A, a member of the TGF- superfamily was thought to be involved in the pathogenesis of UF by increasing the ECM components, such as versican, fibronectin, and collagen 1A1, at least in part through activating the Smad-2/3 and p38-MAPK signaling pathway (Islam et al., 2014

Stimulation of angiogenesis and collagen deposition

BPC-157 reversed corticosteroid-induced muscle damage in preclinical studies, restoring structure and function.- TB-500 showed potential in supporting cardiomyocyte survival and angiogenesis in ischemic models, with evidence of integrin-linked kinase (ILK) and Akt pathway involvement.- Early evidence suggests both compounds may support musculoskeletal healing and bone remodeling