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While current research on combination therapies predominantly focuses on downstream therapeutic biomarkerssuch as pro-inflammatory cytokines (e.g., TNF-, IL-1, IL-6), oxidative stress markers (e.g., MDA, SOD), and mucosal healing indicators (e.g., MUC-2, tight junction proteins)investigations into upstream signaling pathways (e.g., Notch, MAPK/ERK) remain preliminary

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Phosphorylated IRF3 induces Type I interferon synthesis, while STING accelerates NF-B activation through IB phosphorylation (Zhang and Zhang, 2025)