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Likewise, alteration in glutathione metabolism may promote oxidative stress and an inflammatory milieu in LC patients as reported in acute COVID-19 disease (76)

These gaps underscore the need for future research to move beyond descriptive associations, toward mechanistic dissection of pathway integration, cell type-specific functions, and clinical validation of biomarkers and targeted therapies

Caloric restriction and reduced GH/IGF-I signaling improve resistance to cellular stress and inhibit mTOR, thereby increasing longevity and possibly enhancing tumor resistance

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