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Nitric oxide (NO) pathway enhancement: BPC-157 increases endothelial nitric oxide synthase (eNOS) expression and activity

10.2217/nnm-2017-0151 252 SawickaB.MessaoudiM.AcharR

However, sustained suppression of FAO along with persistent enhancement of glycolysis can result in intracellular lipid accumulation, activation of inflammatory pathways, and fibrogenesis, collectively contributing to the progression of AKI (Figure 3)