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Importantly, although A is generally localized toward the synapses, evidence from AD and Downs syndrome (DS) with early AD symptoms exhibit intraneuronal A 142 accumulation that precedes Tau pathology and extracellular A plaque formation (Takahashi et al., 2017

While various therapeutic modalities, including endocrine therapy, immunotherapy, radiation, targeted therapy, and chemotherapy, are available for breast cancer treatment (Dong et al., 2024), interventions specifically targeting mitochondria to induce ferroptosis remain underexplored

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This conversion directly supports mitochondrial bioenergetics, leading to higher adenosine triphosphate (ATP) levels in hepatocytes (liver cells) and preventing the mitochondrial dysfunction that often accompanies severe toxic burden